Angiotensin II Decreases Endothelial Nitric Oxide Synthase Phosphorylation via AT1R Nox/ROS/PP2A Pathway

Angiotensin II Decreases Endothelial Nitric Oxide Synthase Phosphorylation via AT1R Nox/ROS/PP2A Pathway

Increasing evidences suggest that angiotensin (Ang) II participates in the pathogenesis of endothelial dysfunction (ED) through multiple signaling pathways, including angiotensin type 1 receptor (AT1R) mediated NADPH oxidase (Nox)/reactive oxygen species (ROS) signal transduction. However, the detai...

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Bibliographic Details
Main Authors: Jing Ding, Min Yu, Juncai Jiang, Yanbei Luo, Qian Zhang, Shengnan Wang, Fei Yang, Alei Wang, Lingxiao Wang, Mei Zhuang, Shan Wu, Qifang Zhang, Yong Xia, Deqin Lu
Format: Article
Language:English
Published: Frontiers Media S.A. 2020-09-01
Series:Frontiers in Physiology
Subjects:
angiotensin II
angiotensin II type 1 receptor
protein phosphatase 2A
endothelial nitric oxide synthase
NADPH oxidase
Online Access:https://www.frontiersin.org/article/10.3389/fphys.2020.566410/full

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https://www.frontiersin.org/article/10.3389/fphys.2020.566410/full

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