Apolipoprotein A-I Reduces In-Stent Restenosis and Platelet Activation and Alters Neointimal Cellular Phenotype

Apolipoprotein A-I Reduces In-Stent Restenosis and Platelet Activation and Alters Neointimal Cellular Phenotype

Summary: Even the most advanced drug-eluting stents evoke unresolved issues, including chronic inflammation, late thrombosis, and neoatherosclerosis. This highlights the need for novel strategies that improve stent biocompatibility. Our studies show that apolipoprotein A-I (apoA-I) reduces in-stent...

Full description

Bibliographic Details
Main Authors: Laura Z. Vanags, PhD, Joanne T.M. Tan, PhD, Keyvan K. Galougahi, MD, PhD, Andreas Schaefer, MD, Steven G. Wise, PhD, Andrew Murphy, PhD, Ziad A. Ali, MD, PhD, Christina A. Bursill, PhD
Format: Article
Language:English
Published: Elsevier 2018-04-01
Series:JACC: Basic to Translational Science
Online Access:http://www.sciencedirect.com/science/article/pii/S2452302X17302905

Internet

http://www.sciencedirect.com/science/article/pii/S2452302X17302905

Similar Items