Postprandial apoE isoform and conformational changes associated with VLDL lipolysis products modulate monocyte inflammation.

Postprandial hyperlipemia, characterized by increased circulating very low-density lipoproteins (VLDL) and circulating lipopolysaccharide (LPS), has been proposed as a mechanism of vascular injury. Our goal was to examine the interactions between postprandial lipoproteins, LPS, and apoE3 and apoE4 o...

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Bibliographic Details
Main Authors: Laura J den Hartigh, Robin Altman, Romobia Hutchinson, Jitka Petrlova, Madhu S Budamagunta, Sarada D Tetali, Jens O Lagerstedt, John C Voss, John C Rutledge
Format: Article
Language:English
Published: Public Library of Science (PLoS) 2012-01-01
Series:PLoS ONE
Online Access:http://europepmc.org/articles/PMC3509065?pdf=render