Hepatic Knockdown of Endothelin Type A Receptor (ETAR) Ameliorates Hepatic Insulin Resistance and Hyperglycemia Through Suppressing p66Shc-Mediated Mitochondrial Fragmentation in High-Fat Diet-Fed Mice

Hepatic Knockdown of Endothelin Type A Receptor (ETAR) Ameliorates Hepatic Insulin Resistance and Hyperglycemia Through Suppressing p66Shc-Mediated Mitochondrial Fragmentation in High-Fat Diet-Fed Mice

Li Feng,* Songhua Wang,* Feng Chen,* Cheng Zhang, Qiao Wang, Yuting Zhao, Zifeng Zhang School of Life Science, Jiangsu Normal University, Xuzhou, Jiangsu, 221116, People’s Republic of China*These authors contributed equally to this workCorrespondence: Zifeng Zhang 101 Shanghai Road, Xuzhou...

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Bibliographic Details
Main Authors: Feng L, Wang S, Chen F, Zhang C, Wang Q, Zhao Y, Zhang Z
Format: Article
Language:English
Published: Dove Medical Press 2021-03-01
Series:Diabetes, Metabolic Syndrome and Obesity : Targets and Therapy
Subjects:
endothelin-1
endothelin type a receptor
66-kda src homology 2 domain-containing protein
mitochondrial fragmentation
metabolism abnormality
Online Access:https://www.dovepress.com/hepatic-knockdown-of-endothelin-type-a-receptor-etar-ameliorates-hepat-peer-reviewed-article-DMSO

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https://www.dovepress.com/hepatic-knockdown-of-endothelin-type-a-receptor-etar-ameliorates-hepat-peer-reviewed-article-DMSO

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