Overexpression of PEP-19 Suppresses Angiotensin II–Induced Cardiomyocyte Hypertrophy

Overexpression of PEP-19 Suppresses Angiotensin II–Induced Cardiomyocyte Hypertrophy

Abstract.: The precise molecular mechanisms leading to disturbance of Ca2+/calmodulin-dependent intracellular signalling in cardiac hypertrophy remains unclear. As an endogenous calmodulin regulator protein, the pathophysiology role of PEP-19 during cardiac hypertrophy was investigated in the presen...

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Bibliographic Details
Main Authors: Yang-yang Xie, Meng-meng Sun, Xue-fang Lou, Chen Zhang, Feng Han, Bo-ya Zhang, Ping Wang, Ying-mei Lu
Format: Article
Language:English
Published: Elsevier 2014-01-01
Series:Journal of Pharmacological Sciences
Online Access:http://www.sciencedirect.com/science/article/pii/S1347861319301215

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http://www.sciencedirect.com/science/article/pii/S1347861319301215

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