Persistent Reversal of Enhanced Amphetamine Intake by Transient CaMKII Inhibition

Persistent Reversal of Enhanced Amphetamine Intake by Transient CaMKII Inhibition

Amphetamine exposure transiently increases Ca2+/calmodulin-dependent protein kinase II (CaMKII) α expression in the nucleus accumbens (NAcc) shell and this persistently increases local GluA1 S831 phosphorylation and enhances behavioral responding to the drug. Here we assessed whether transiently int...

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Main Authors: Loweth, Jessica A. (Author), Li, Dongdong (Author), Cortright, James J. (Author), Wilke, Georgia (Author), Jeyifous, Okunola (Author), Neve, Rachael L. (Contributor), Bayer, K. Ulrich (Author), Vezina, Paul (Author)
Other Authors: Massachusetts Institute of Technology. Department of Brain and Cognitive Sciences (Contributor)
Format: Article
Language:English
Published: Society for Neuroscience, 2013-08-01T18:53:27Z.
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