A Cdx4-Sall4 Regulatory Module Controls the Transition from Mesoderm Formation to Embryonic Hematopoiesis

A Cdx4-Sall4 Regulatory Module Controls the Transition from Mesoderm Formation to Embryonic Hematopoiesis

Deletion of caudal/cdx genes alters hox gene expression and causes defects in posterior tissues and hematopoiesis. Yet, the defects in hox gene expression only partially explain these phenotypes. To gain deeper insight into Cdx4 function, we performed chromatin immunoprecipitation sequencing (ChIP-s...

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Bibliographic Details
Main Authors: Paik, Elizabeth J (Author), Mahony, Shaun (Author), White, Richard M (Author), Price, Emily N (Author), DiBiase, Anthony (Author), Dorjsuren, Bilguujin (Author), Mosimann, Christian (Author), Davidson, Alan J (Author), Zon, Leonard I (Author), Gifford, David K. (Contributor)
Other Authors: Massachusetts Institute of Technology. Computer Science and Artificial Intelligence Laboratory (Contributor), Massachusetts Institute of Technology. Department of Electrical Engineering and Computer Science (Contributor)
Format: Article
Language:English
Published: Elsevier, 2014-10-02T17:42:53Z.
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