Histone Deacetylase 6 (HDAC6) Is Critical for Tumor Cell Survival and Promotes the Pro-Survival Activity of 14-3-3ζ viaDeacetylation of Lysines Within the14-3-3ζ Binding Pocket
Our understanding of non-histone acetylation as a means of cellular regulation is in its infancy. Using a mass spectrometry approach we identified acetylated lysine residues and monitored acetylation changes across the proteome as a consequence of metabolic stress (hypoxia). We observed changes in a...
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| Format: | Others |
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BYU ScholarsArchive
2015
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| Online Access: | https://scholarsarchive.byu.edu/etd/5568 https://scholarsarchive.byu.edu/cgi/viewcontent.cgi?article=6567&context=etd |