The Activation of Novel Calcium-dependent Pathways Downstream of N-methyl-D-aspartate Receptors
Calcium (Ca2+) influx through N-methyl-D-asparate receptors (NMDARs) is widely held to be the requisite step initiating delayed neuronal death following ischemic stroke. However, blocking NMDARs fails to prevent the accumulation of intracellular Ca2+ ([Ca2+]i) and subsequent neurotoxicity. This su...
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| Language: | en_ca |
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2009
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| Online Access: | http://hdl.handle.net/1807/24324 |