FK866 reduces axon pathology in in vitro and in vivo models of Huntington's disease and Alzheimer's disease and attenuates behavioral abnormalities
Among its various biological functions, NAD metabolism regulates axon degeneration, which is an early and often causative event in a variety of neurodegenerative diseases including Huntington’s disease (HD) and Alzheimer’s disease (AD). Our research group previously found that reducing the levels of...
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University of Nottingham
2016
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| Online Access: | http://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.689820 |