Personalised therapy based on immunopathogenesis to improve outcome of at-risk and established systemic lupus erythematosus
Background: Pathogenesis of systemic lupus erythematosus (SLE) is thought to be closely related to B-cell dysfunction, and accordingly this is the usual target for therapies. However, non-B-cell mechanisms such as tumour necrosis factor (TNF) and interferons may also be important in the onset as wel...
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University of Leeds
2018
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| Online Access: | https://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.759814 |