Smad6 Methylation Represses NFκB Activation and Periodontal Inflammation

Smad6 Methylation Represses NFκB Activation and Periodontal Inflammation

The balance between pro- and anti-inflammatory signals maintains tissue homeostasis and defines the outcome of chronic inflammatory diseases such as periodontitis, a condition that afflicts the tooth-supporting tissues and exerts an impact on systemic health. The induction of tissue inflammation rel...

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Bibliographic Details
Main Authors: Chai, Y. (Author), Feng, J. (Author), Gou, Y. (Author), Hajishengallis, G. (Author), Ho, T.V (Author), Jackson-Weaver, O. (Author), Jin, J. (Author), Liu, J. (Author), Richard, S. (Author), Shen, Y. (Author), Ungvijanpunya, N. (Author), Wu, J. (Author), Xu, J. (Author), Zhang, T. (Author)
Format: Article
Language:English
Published: SAGE Publications Inc. 2018
Subjects:
arginine
Arginine
arginine methylation
cell culture
cell signaling
Cells, Cultured
cytology
gingiva
Gingiva
human
Humans
immunoglobulin enhancer binding protein
immunology
inflammation
Inflammation
methylation
Methylation
MYD88 protein, human
myeloid differentiation factor 88
Myeloid Differentiation Factor 88
NF-kappa B
periodontal disease
periodontitis
Periodontitis
post-translational modification
PRMT1 protein, human
protein arginine methyltransferase
protein domain
Protein Interaction Domains and Motifs
Protein-Arginine N-Methyltransferases
protein-protein interaction
repressor protein
Repressor Proteins
signal transduction
Signal Transduction
Smad6 protein
Smad6 Protein
SMAD6 protein, human
SMURF1 protein, human
transforming growth factor beta
Transforming Growth Factor beta
ubiquitin protein ligase
Ubiquitin-Protein Ligases
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