Antonio Vidal-Puig

Antonio Vidal-Puig, Spanish scientist Antonio Vidal-Puig (born Valencia, Spain June 12, 1962) is a Spanish medical doctor and scientist who works as a Professor of Molecular Nutrition and Metabolism at the University of Cambridge (UK), best known for advancing the concept that pharmacological targeting of brown fat may serve to treat overweight and obesity in affected individuals, as well as for introducing the concept of adipose tissue "expandability" as an important factor in the pathogenesis of insulin resistance in the context of positive energy balance. His published work focuses on areas such as adipose tissue metabolism and lipotoxicity, regulation of insulin secretion,  and the pathophysiology of metabolic syndrome, obesity, and type 2 diabetes. Provided by Wikipedia
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  1. 1
    Studying Brown Adipose Tissue in a Human in vitro Context
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  2. 2
    Nothing Iffy about HIF in the Hypothalamus.
    by Sam Virtue, Antonio Vidal-Puig
    Published 2011-07-01
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  3. 3
    It's not how fat you are, it's what you do with it that counts.
    by Samuel Virtue, Antonio Vidal-Puig
    Published 2008-09-01
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  4. 4
    PPARs and Metabolic Disorders Associated with Challenged Adipose Tissue Plasticity
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  5. 5
    Stearoyl-CoA Desaturase 1 Is a Key Determinant of Membrane Lipid Composition in 3T3-L1 Adipocytes.
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  6. 6
    Editorial: Brown Adipose Tissue: From Heat Production in Rodents to Metabolic Health in Humans
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  7. 7
    Genome-wide profiling of microRNAs in adipose mesenchymal stem cell differentiation and mouse models of obesity.
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  8. 8
    The Obese Species: a special issue on obesity and metabolic disorders
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  9. 9
    Metabolomic and Lipidomic Analysis of the Heart of Peroxisome Proliferator-Activated Receptor-γ Coactivator 1-β Knock Out Mice on a High Fat Diet
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  10. 10
    Brown Adipose Tissue Thermogenic Capacity Is Regulated by Elovl6
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  11. 11
    Surplus fat rapidly increases fat oxidation and insulin resistance in lipodystrophic mice
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  12. 12
    Calcium Channel CaV2.3 Subunits Regulate Hepatic Glucose Production by Modulating Leptin-Induced Excitation of Arcuate Pro-opiomelanocortin Neurons
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  13. 13
    Olanzapine-induced hyperphagia and weight gain associate with orexigenic hypothalamic neuropeptide signaling without concomitant AMPK phosphorylation.
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  14. 14
    Macrophage beta2-adrenergic receptor is dispensable for the adipose tissue inflammation and function
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  15. 15
    Publisher Correction: Enhanced β-adrenergic signalling underlies an age-dependent beneficial metabolic effect of PI3K p110α inactivation in adipose tissue
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  16. 16
    Enhanced β-adrenergic signalling underlies an age-dependent beneficial metabolic effect of PI3K p110α inactivation in adipose tissue
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  17. 17
    Accelerated phosphatidylcholine turnover in macrophages promotes adipose tissue inflammation in obesity
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  18. 18
    Mitochondrial fusion is increased by the nuclear coactivator PGC-1beta.
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  19. 19
    Autocrine IGF2 programmes β-cell plasticity under conditions of increased metabolic demand
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  20. 20
    Stress-induced activation of brown adipose tissue prevents obesity in conditions of low adaptive thermogenesis
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