Santiago Ramón y Cajal

Ramón y Cajal in 1899 Santiago Ramón y Cajal (; 1 May 1852 – 17 October 1934) was a Spanish neuroscientist, pathologist, and histologist specializing in neuroanatomy and the central nervous system. He and Camillo Golgi received the Nobel Prize in Physiology or Medicine in 1906. Ramón y Cajal was the first person of Spanish origin to win a scientific Nobel Prize. His original investigations of the microscopic structure of the brain made him a pioneer of modern neuroscience.

Hundreds of his drawings illustrating the arborization (tree-like growth) of brain cells are still in use, since the mid-20th century, for educational and training purposes. Provided by Wikipedia
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  1. 1
    La Psicología de Don Quijote de la Mancha y el Quijotismo
    by Santiago Ramón y Cajal
    Published 2004-09-01
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  2. 2
    Interplay Between ncRNAs and Cellular Communication: A Proposal for Understanding Cell-Specific Signaling Pathways
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  3. 3
    Spontaneous Cell Detachment and Reattachment in Cancer Cell Lines: An In Vitro Model of Metastasis and Malignancy
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  4. 4
    Epithelial-mesenchymal transition markers and HER3 expression are predictors of elisidepsin treatment response in breast and pancreatic cancer cell lines.
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  5. 5
    RPLP1, a crucial ribosomal protein for embryonic development of the nervous system.
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  6. 6
    Phosphorylation of eIF4E Confers Resistance to Cellular Stress and DNA-Damaging Agents through an Interaction with 4E-T: A Rationale for Novel Therapeutic Approaches.
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  7. 7
    Ovarian Fibrosarcoma: Clinicopathologic Considerations about the Intraoperative and Post-Surgical Procedures
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  8. 8
    Brain ApoA-I, ApoJ and ApoE Immunodetection in Cerebral Amyloid Angiopathy
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  9. 9
    Publisher Correction: ITGB3-mediated uptake of small extracellular vesicles facilitates intercellular communication in breast cancer cells
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  10. 10
    ITGB3-mediated uptake of small extracellular vesicles facilitates intercellular communication in breast cancer cells
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  11. 11
    Cx43 and Associated Cell Signaling Pathways Regulate Tunneling Nanotubes in Breast Cancer Cells
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  12. 12
    Insight into the Role and Regulation of Gap Junction Genes in Lung Cancer and Identification of Nuclear Cx43 as a Putative Biomarker of Poor Prognosis
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  13. 13
    Validation of Cell-Free DNA Collection Tubes for Determination of EGFR Mutation Status in Liquid Biopsy from NSCLC Patients
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  14. 14
    Sequential immunohistochemistry and virtual image reconstruction using a single slide for quantitative KI67 measurement in breast cancer
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  15. 15
    miR-125b acts as a tumor suppressor in breast tumorigenesis via its novel direct targets ENPEP, CK2-α, CCNJ, and MEGF9.
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  16. 16
    Dissecting Breast Cancer Circulating Tumor Cells Competence via Modelling Metastasis in Zebrafish
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  17. 17
    Fluorescence in situ hybridization and immunohistochemistry as diagnostic methods for ALK positive non-small cell lung cancer patients.
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  18. 18
    Balance between MKK6 and MKK3 mediates p38 MAPK associated resistance to cisplatin in NSCLC.
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  19. 19
    Characterizing the Invasive Tumor Front of Aggressive Uterine Adenocarcinoma and Leiomyosarcoma
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  20. 20
    Five microRNAs in Serum Are Able to Differentiate Breast Cancer Patients From Healthy Individuals
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