The HIV matrix protein p17 subverts nuclear receptors expression and induces a STAT1-dependent proinflammatory phenotype in monocytes.

The HIV matrix protein p17 subverts nuclear receptors expression and induces a STAT1-dependent proinflammatory phenotype in monocytes.

BACKGROUND: Long-term remission of HIV-1 disease can be readily achieved by combinations of highly effective antiretroviral therapy (HAART). However, a residual persistent immune activation caused by circulating non infectious particles or viral proteins is observed under HAART and might contribute...

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Bibliographic Details
Main Authors: Barbara Renga, Daniela Francisci, Claudio D'Amore, Elisabetta Schiaroli, Andrea Mencarelli, Sabrina Cipriani, Franco Baldelli, Stefano Fiorucci
Format: Article
Language:English
Published: Public Library of Science (PLoS) 2012-01-01
Series:PLoS ONE
Online Access:http://europepmc.org/articles/PMC3340403?pdf=render

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http://europepmc.org/articles/PMC3340403?pdf=render

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