Cigarette smoke modifies neutrophil chemotaxis, neutrophil extracellular trap formation and inflammatory response-related gene expression

Cigarette smoke modifies neutrophil chemotaxis, neutrophil extracellular trap formation and inflammatory response-related gene expression

Background and Objective: Cigarette smoking is a major risk factor for periodontitis, and smoking perturbs neutrophil reactive oxygen species production. This study tested the hypothesis that cigarette smoke extract (CSE) and its components/metabolites nicotine, cotinine and thiocyanate (SCN-), may...

Full description

Bibliographic Details
Main Authors: Chapple, I.L.C (Author), Cooper, P.R (Author), Hirschfeld, J. (Author), Matthews, J.B (Author), Milward, M.R (Author), White, P.C (Author), Wright, H.J (Author)
Format: Article
Language:English
Published: Blackwell Munksgaard 2018
Subjects:
apoptosis
Apoptosis
chemotaxis
Chemotaxis
cigarette smoke
cotinine
Cotinine
drug effect
extracellular trap
Extracellular Traps
flow cytometry
Flow Cytometry
gene expression
Gene Expression
human
Humans
inflammation
metabolism
neutrophil
neutrophils
Neutrophils
nicotine
Nicotine
periodontitis
reactive oxygen metabolite
Reactive Oxygen Species
real time polymerase chain reaction
Real-Time Polymerase Chain Reaction
smoke
Smoke
smoking
Smoking
thiocyanate
Thiocyanates
thiocyanic acid derivative
Online Access:View Fulltext in Publisher

Internet

View Fulltext in Publisher

Similar Items