Cigarette smoke modifies neutrophil chemotaxis, neutrophil extracellular trap formation and inflammatory response-related gene expression
Background and Objective: Cigarette smoking is a major risk factor for periodontitis, and smoking perturbs neutrophil reactive oxygen species production. This study tested the hypothesis that cigarette smoke extract (CSE) and its components/metabolites nicotine, cotinine and thiocyanate (SCN-), may...
Main Authors: | Chapple, I.L.C (Author), Cooper, P.R (Author), Hirschfeld, J. (Author), Matthews, J.B (Author), Milward, M.R (Author), White, P.C (Author), Wright, H.J (Author) |
---|---|
Format: | Article |
Language: | English |
Published: |
Blackwell Munksgaard
2018
|
Subjects: | |
Online Access: | View Fulltext in Publisher |
Similar Items
-
Association between Cigarette Smoking and New-Onset Diabetes Mellitus in 78,212 Koreans Using Self-Reported Questionnaire and Urine Cotinine
by: Ji Hye Kim, et al.
Published: (2020-06-01) -
Effect of smoking on neutrophil apoptosis in chronic periodontitis: An immunohistochemical study
by: Sachin S Shivanaikar, et al.
Published: (2013-01-01) -
Assessment of smoking status based on cotinine levels in nasal lavage fluid
by: Mehmet Hakan Ozdener, et al.
Published: (2009-07-01) -
The health effects of menthol cigarettes as compared to non-menthol cigarettes
by: Allison C. Hoffman
Published: (2011-05-01) -
Genetics of Tobacco Use
by: Nancy Nairi Maserejian, et al.
Published: (2004-06-01)