Marie Celeste Simon

Marie Celeste Simon is the Arthur H. Rubenstein Professor of cell and developmental biology, the scientific director of the Abramson Family Cancer Research Institute, and the associate director of the Abramson Cancer Center Core Facilities, at the University of Pennsylvania Perelman School of Medicine. Her research focuses on cancer cell metabolism, angiogenesis, and immunology.

Simon earned a B.A. from Miami University in 1977, and a M.S. from Ohio State University in 1980, both in microbiology. She earned a Ph.D. in molecular biology from The Rockefeller University in 1985. She did postdoctoral research with Joseph Nevins at Rockefeller, and with Stuart Orkin at Harvard Medical School.

In 1992, Simon became an Assistant Professor of Medicine and Molecular Genetics and Cell Biology at the University of Chicago. She became a professor at the University of Pennsylvania School of Medicine in 1999.

Simon was an HHMI investigator from 1994 to 2014. In 2017, she was awarded the National Cancer Institute Outstanding Investigator Award. She was elected to the National Academy of Medicine in 2018. In 2021, she was elected member of the U. S. National Academy of Sciences. Provided by Wikipedia
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  1. 1
    Hidden features: exploring the non-canonical functions of metabolic enzymes
    by Peiwei Huangyang, M. Celeste Simon
    Published 2018-08-01
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  2. 2
    Cellular adaptation to oxygen deficiency beyond the Nobel award
    by José López-Barneo, M. Celeste Simon
    Published 2020-01-01
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  3. 3
    Publisher Correction: Cellular adaptation to oxygen deficiency beyond the Nobel award
    by José López-Barneo, M. Celeste Simon
    Published 2020-02-01
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  4. 4
    PIK3R3, part of the regulatory domain of PI3K, is upregulated in sarcoma stem-like cells and promotes invasion, migration, and chemotherapy resistance
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  5. 5
    PPARγ is dispensable for clear cell renal cell carcinoma progression
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  6. 6
    PI3K/Akt pathway and Nanog maintain cancer stem cells in sarcomas
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  7. 7
    E2A Antagonizes PU.1 Activity through Inhibition of DNA Binding
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  8. 8
    Myeloid Cell Hypoxia-Inducible Factors Promote Resolution of Inflammation in Experimental Colitis
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  9. 9
    Epigenetic re-expression of HIF-2α suppresses soft tissue sarcoma growth
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  10. 10
    Triglycerides Promote Lipid Homeostasis during Hypoxic Stress by Balancing Fatty Acid Saturation
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  11. 11
    Targeting glutamine metabolism slows soft tissue sarcoma growth
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  12. 12
    Nontranscriptional Role of Hif-1α in Activation of γ-Secretase and Notch Signaling in Breast Cancer
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  13. 13
    DNA methylation repels binding of hypoxia-inducible transcription factors to maintain tumor immunotolerance
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